Cellular changes in fat tissue -- not the immune system -- lead to the "hyperinflammation" characteristic of obesity-related glucose intolerance and type 2 diabetes, according to new research from the University of Cincinnati (UC)...That means lose the weight, fatsos.
"This finding is quite novel because current drug development efforts target immune cells (macrophages, T-cells) to eliminate this hyperinflammation. Our research suggests obesity-related glucose intolerance has nothing to do with the immune system. It may be more effective to target adipocytes (fat cells)," explains Moscat, principal investigator of the study and chair of UC's cancer and cell biology department.
Sunday, July 18, 2010
Fat Cells Play Key Role in Development of Type 2 Diabetes
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